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Additionally, PD pathogenesis primarily involves vesicular transport and the maintenance of lysosomes and mitochondria [50]

Each route produces different absorption kinetics, so alternating between them creates inconsistent plasma levels that confound data interpretation

[205] [198] [76] Phosphorylation of DAT by either kinase induces transporter internalization (non-competitive reuptake inhibition), but PKC-mediated phosphorylation alone induces the reversal of dopamine transport through DAT (i.e., dopamine efflux)